Heart attacks kill almost 700,000 men and women each year in the United States. Signs of a heart attack usually include chest discomfort. Symptoms of a Heart Attack can be different in a man than the heart attack symptoms in a woman. Heart attacks should be suspected if you experience pressure or pain in your chest, shortness of breath, nausea, dizziness or anxiety.

Duration : 0:6:11

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Technorati Tags: asktheholisticdoctor, attack, dahlman, drdahlman, health, heart, symptoms

Preventing Heart Disease and Heart Attack Educational Video. U.S. Department of Health and Human Services National Institutes of Health; National Heart, Lung, and Blood Institute; Act in Time to Heart Attack Signs; Item #56-042N, September 2001; The dramatic, moving stories of three heart attack survivors and their families illustrate the importance of heeding heart attack warning signs and seeking medical care quickly. They vividly convey how a real heart attack may differ from the stereotypical “movie heart attack” and how getting immediate treatment can save lives. The warm and sympathetic narration by an emergency department physician explains what a heart attack is, the treatments that can save lives if given quickly, why many heart attack victims delay seeking care, and how to make a heart attack survival plan. Useful for health fairs, medical waiting rooms, community groups, and home viewing. Producer: National Institutes of Health; Keywords: hhs.gov; public_safety; Creative Commons license: Public Domain. Heart Attack Warning Signs. A heart attack is a frightening event, and you probably don’t want to think about it. But, if you learn the signs of a heart attack and what steps to take, you can save a life–maybe your own. What are the signs of a heart attack? Many people think a heart attack is sudden and intense, like a “movie” heart attack, where a person clutches his or her chest and falls over. The truth is that many heart attacks start slowly, as a mild pain or discomfort. If you feel such a symptom, you may not be sure what’s wrong. Your symptoms may even come and go. Even those who have had a heart attack may not recognize their symptoms, because the next attack can have entirely different ones. Women may not think they’re at risk of having a heart attack–but they are. Learn more about women and heart attack. It’s vital that everyone learn the warning signs of a heart attack. These are: Chest discomfort. Most heart attacks involve discomfort in the center of the chest that lasts for more than a few minutes, or goes away and comes back. The discomfort can feel like uncomfortable pressure, squeezing, fullness, or pain. Discomfort in other areas of the upper body. Can include pain or discomfort in one or both arms, the back, neck, jaw, or stomach. Shortness of breath. Often comes along with chest discomfort. But it also can occur before chest discomfort. Other symptoms. May include breaking out in a cold sweat, nausea, or light-headedness. Learn the signs–but also remember: Even if you’re not sure it’s a heart attack, you should still have it checked out. Fast action can save lives-maybe your own. After you learn more about Heart Attack, try a brief quiz to see if you know what to do if you or someone else has warning signs. How do you survive a heart attack? Fast action is your best weapon against a heart attack. Why? Because clot-busting drugs and other artery-opening treatments can stop a heart attack in its tracks. They can prevent or limit damage to the heart–but they need to be given immediately after symptoms begin. The sooner they are started, the more good they will do–and the greater the chances are for survival and a full recovery. To be most effective, they need to be given ideally within 1 hour of the start of heart attack symptoms. You can reduce your risk of having a heart attack—even if you already have coronary Heart Disease (CHD) or have had a previous heart attack. The key is to take steps to prevent or control your heart disease risk factors. Six Key Steps To Reduce Heart Attack Risk; Taking these steps will reduce your risk of having a heart attack: Stop smoking; Lower high blood pressure; Reduce high blood cholesterol; Aim for a healthy weight; Be physically active each day. Manage diabetes.

Duration : 0:13:12

Technorati Tags: Angina, arrhythmia, arterties, artery, attack, cardiologist, coronary, disease, diseases, doctor, failure, hardening, heart, symptons

Heart Disease affects more than 20 million Americans. Let’s explore the various forms that heart disease can take.Watch More Health Videos at Health Guru: http://www.healthguru.com/?YT

Duration : 0:2:55

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Visit http://cardiology.healthanimations.com for FREE videos, illustrations and patient handouts for healthcare providers. This 3d medical animation shows a Heart Attack resulting from atherosclerosis, which is inflammation of an artery. It was designed to be an immersive experience to hold the viewer’s interest in this complex, slow-developing process. Every shot was conceptualized as a visual effect to evoke the sensation of a journey within a dynamic and diverse environment. Beginning with the probable cause, which is damage to the endothelial wall, the story progresses in detail through the stages of atherosclerosis concluding with the resulting damage to the heart. See more 3D medical animations from Nucleus Medical Art at http://www.nucleusinc.com/youtube ANCE00192

Duration : 0:2:52

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The muscular scheme is critical to the humane body. It is essential from the point of view that it helps in movement. One can’t imagine the aftermaths if we could move or grow! We would, in all prospect wouldn’t have existed in the primary place. The muscle tissues in addition in making us move are even more important due to the following characteristics.

1. Excitability: What is our normal reaction when we touch a hot plate? We without delay withdraw the portion in contact with the hot object. This is known as stimulus and the humane body is programmed to respond to stimuli. The muscle tissues in our body send signals to the brain and the brain tells the body to move out – all this happens within a fraction of a second! Had this taken any longer, we would have burnt our hands, till the time our brain told us to remove the hand!

All this and more is possible because of the excitability function of the muscle tissues.

2. Contractility: In simple words, our muscles may contract. One might question the need for our muscles to contract. If our muscles wouldn’t contract, then we wouldn’t be competent to lift things or sit. The folding of our muscles is genuinely contraction taking place and this is the body’s distinguishable self-accommodating scheme that helps the muscles be contained within the same frame rather than tearing out for space!

Also, due to this contraction, a power is produced in the muscles which give the energy to carry out strenuous activities.

3. Extensibility: This is the opposite of the former function. There has to be a similar opposite action in nature and that is best shown by this muscle functioning. After the muscle contracts, it ought to extend to release the energy which it had stored while contraction.

Think of it as when we stretch and when we pull things – without muscle extension, it would not have been possible. Also, extension of the muscles helps growth and is somewhat indirectly related to control of the body temperature (both contraction and extension in pair).

4. Elasticity: If our muscles weren’t elastic, we would have been longer and taller with each extension! Think of the phenomena as a rubber band – it is elastic to a sure limit, beyond which, it breaks. Similarly, our muscles stretch to a sure limit without injury and then come back to it is primary shape and size. Elasticity of muscle fibers is exceedingly important.

5. Conductivity: The muscle tissues in our body conduct ion interchange and aid carry electrical impulses from the brain and nerves to the body parts and vice-versa. This is achieved by the muscles preserving a steady electrical field which facilitates the motion of charged ions.

MMA workouts will help maintain the above brought up characteristics of the muscle tissues and keep you in good health.

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In medical terminology ‘Heart Surgery’ refers to a surgical routine which intends to replace the blocked coronary arteries with the veins received from the lower extremities of a patient. The usual procedure involves placing the patient on a heart bypass machine. Then the surgeon performs surgery on the non-beating resting heart of the patient. With the surgery, it unquestionably improves the quality of life of a heart ailment sufferer.

In order to repair dissimilar heart difficultnesses of a patient, medical science has come up with some types of heart surgeries. Below is the brief description of these heart surgeries:

Coronary Artery Bypass Grafting: It is also abbreviated as CABG and is one of the most mutual types of heart surgeries. CABG is normally carried on a person with a serous coronary artery sickness (CAD).

In such type of coronary artery disease, a fatty material referred to as ‘Plaque’ gets collected inside the coronary arteries of a patient, which then blocks the arteries and reduces the flow of blood in the heart muscle.

In this type of heart surgery, a surgeon ordinarily takes an artery or a vein from the leg, chest or any other body portion of a patient. He then grafts or connects them (veins /arteries) with the blocked artery. With this, the CABG helps to bypass the blockage. In one surgery, the surgeon may bypass the greatest or most complete or best possible of four blocked coronary arteries.

Transmyocardial Laser Revascularization (TLR): This type of heart surgery is commonly carried out to treat angina. This is the last option of heart surgery when all the other treatment choices have failed.

In this type of heart surgery, the surgeon efficaciously uses the laser technology to make channels in the heart muscle. These channels in turn concede the blood to flow directly from the heart chambers into the heart muscle.

Valve Repair /Replacement: Heart valves are responsible for letting blood flow in one direction. Each valve comprises of a set of flaps known as ‘Leaflets’.

These leaflets when get opened grant the blood to pass directly into the arteries from heart chambers. Thereafter, the leaflets get closed with resolute determination to stop blood from flowing back and forth into the heart chambers.

Valve fix is a kind of heart surgery which aims at operating the closed leaflets. The leaflets get closed due to the valve fusion which in the long run results in the flow of blood from the valves into the arteries getting discontinue.

To win a victory over this problem, surgeons normally carry out valve repair or replacement. These substitute valves incorporate of human, animal tissue or any man-made substance.

Aneurysm Repair: An aneurysm refers to the unnatural bulging in the heart muscle or in the walls of the artery. This trouble normally occurs in the left-most heart chamber. With the passage of time, aneurysm may grow worst and gets burst, therefore leading to the bleeding inside the body. It may also result in heart attack.

In order to win a victory over this trouble, surgeons normally carry out a surgery to replace the weaker sections of the artery with a graft.

Heart Transplant: Heart Transplantation is normally done on those people who are in need of medical care who have weak heart that fails to pump sufficient blood for meeting the respective body requirements.

This type of surgery intends to replace the un-healthy and diseased heart with the healthful heart. This surgical measure is normally practiced when all the medical treatments fail.

Only those people who are in need of medical care who are either badly sick or need an instant heart transplantation are commended to carry out this surgical remedy. This is due to the fixed availability of donor hearts.

It is completely up to the medical surgeon who diagnoses your heart problem conservatively to determine which surgical procedure suits you best and then carry out the suitable Heart surgery.

Part Of The Human Heart 2

Slightly enlarged. Section through ventricles and auricles shows cardiac valves. Dissectible into 2 halves. Size, 27 x 12 x 14 cm. On stand with base.

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The female body has a lot of erogenous zones, or hot spots that when sexually stimulated may give her amazing orgasms. Today in Female Sexual Anatomy 101 we’ll learn when it comes to the C-spot, the G-spot and the A-spot. Oh, and even the U-Spot.

The C-spot (Also known as the Fabulous Clitoris)

The clitoris is the number one hot spot on the female pleasure zone and 70% of women get off from clitoral stimulation. The only organ designed solely for pleasure, this little sensible glans is located just beneath where the top of the inner vaginal lips meet.

The clitoris has over 8000 sensible nerve endings, which is more than any other share of the humane body, including the male penis. For this reason, it may be exceedingly sensible until a woman is totally aroused, so start out with foreplay initial on other areas of the body–the lips, the breasts, the vulva–before attacking the clitoris.

While a good deal of women want and need direct stimulation of the clitoris, for other women this may be uncomfortable and even painful. So again, begin slow, use lots of lube, and circle your way around the clitoris before touching the sensible glans directly.

Many women find the top right hand side of the clitoris is very sensible so undertake this spot using a lubricated finger, tongue or vibrator to stimulate her. If her clitoris was a clock, the hot spot would be at 10:30pm. Try focusing on dissimilar time zones around her little clock and watch her body language as she responds. Or ask her to tell you which area feels best to her.

Nerves from the clitoris travel through the vagina, the bladder and urethra, passing along any sensations formulated in that area. This means that clitoral orgasms are almost affiliated to G spot orgasms, even even though they feel much different.

The G Spot (or Infamous G Spot)

The notorious G-spot, or Grafenberg Spot, (also known as the Female Prostate) is talked when it comes to a lot, but for most women it remains elusive. It is an exceedingly sensible area on the front wall inside the vaginal canal approximately one to two inches up, near the bladder. It was firstborn described it as “an (erotic|sexual pleasure|sexually arousing zone located on the anterior wall of the vagina along the course of the urethra that would great for the duration of sexual stimulation.”

That seems easy enough, so why it is so hard to find? The G spot is oftentimes elusive because in it is sexually “un-stimulated” state it doesn’t feel pleasure and it very small. But once aroused, the G spot will commence to outstanding with fluid, and a wrinkly, prune-like tissue (the urethral sponge) will make itself noticeable to touch. It will likewise get started to feel pleasurable to be touched.

Like most forms of sexual arousal, it is better to warm her up original with foreplay, then attempting to search for the G spot right away. Once your lover is aroused, you may then insert two fingers, or a G spot dildo in her vagina to stimulate the G spot. And, clitoral stimulation before and for the duration of G spot arousal works to get the G spot excessively affected emotionally as well.

To stimulate the G spot with your fingers make sure they are well lubricated and your nails are trimmed short (or use a glove and a large total of lube). Insert your fingers 1″-2″ inside the vagina and feel for the puffy, wrinkly sponge-like spot just blow her pelvis. The G spot will feel more or less like the roof of your mouth when sentiment it with your tongue, whereas the surrounding tissue will feel like the insides of your cheeks, very smooth. There you’ve found it.

Start by using a “come hither” motion with your fingers to gently massage the G spot, and add firm pressure when she begins thrusting her hips towards you. The G spot craves firm pressure and thrusting when it is very aroused. A firm, curved dildo or vibrator made for G spot pleasure will likewise work well for this technique.

When a woman is ready to orgasm by way of G spot stimulation she may ejaculate so be prepared. Absorbent towels are good to have handy. The strength of the orgasm may also push your fingers or any toy out of the vagina as well.

The U-Spot. What is it?

The U-Spot is a very little ridge of sensible tissue that is found on either side of the urethral opening and above it. This area is highly (erotic|sexual pleasure|sexually arousing for a lot of women when caressed lightly with a finger, tongue or soft vibrator. The U-spot is highly sensitive, so be very careful when stimulating it and use a great deal of lube. And, like any form of sexual stimulation, ask your collaborator what feels good and try varying speed, pressure and type of stimulation.

Also note, because this area is connected to the urethral (and the female prostate gland), stimulating it may make the woman feel like she needs to urinate. If that is not a sensation she wants to feel, you may want to stay clear from this area. However, together with G spot stimulation, pleasuring the U-spot may be very (erotic|sexual pleasure|sexually arousing and aid coax the ejaculate out when she is in regards to to orgasm.

Bonus Hot Spot- The A-Spot

The A-spot is a sensible patch of tissue at the very end of the vaginal tube above the cervix and beneath the bladder. Also known as the ‘female degenerated prostate’, direct stimulation of this area may construct “violent orgasmic contractions”. Believe me, you will recognise when you hit it.

Pressure on this area gives rise to rapid lubrication of the vagina, even in women who are not normally sexually responsive. This spot may only be hit for the duration of vagina intercourse, or with a long more or less curved dildo. The best position for hitting this spot by way of sex is doggie style.

So, now you recognise all three (actually four) of the most sensible sexual elements of the female body and how to stimulate them. Just effigy if there was a hot spot for each letter in the alphabet? Who knows, perchance there is.

Technorati Tags: a-spot, c-spot, clitoris, erogenous zones, female body, g spot, sensitive sexual parts, sexual parts, u-spot

The omega 3 fatty acids are key constituents in the good functioning of the humane body. The smallest and the most indispensable part of our body, the cells, are surrounded by a cell membrane. This membrane is composed primarily of fatty acids. The cellular membrane has a critical role in the cell’s life, as it allows nutrients into the cell itself. In the same manner, it allows the quick remotion of cell waste products. The cell membrane has to be integer and flexible to permit these substance changes.

If the cell’s membrane loses it is integrity, it will not be capable to hold water and nutrients. The cellular membrane allows for communication amid cells, keeping body organs in good shape. If the membrane begins to lose it is integrity, the cell will not be competent to commune with other cells. Scientists believe that one of the constituents which favor the rapid growth of cancerous tumors is the loss of inter-cellular communication.

There is a close kinship amongst the types of fat we eat and the integrity and fluidity of cell membranes, as the membrane is made up of fats. One of the deviations amid the two types of fats, the omega 3 fatty acids and the saturated/hydrogenated fats is the way they behave at room temperature: the omega 3 fats are liquid in these conditions, while the hydrogenated fats are solid. Due to this reason, if you eat for a long amount of time of time – a lot of completely filled fats, chances are that your cell membrane will be harder and will lack fluidity, whereas a diet rich in omega 3 fatty acids will lead to more liquid cell membranes.

Recent studies have concluded that the omega 3 fatty acids play an crucial role in preventing breast cancer development. The November 2005 edition of the International Journal of Cancer publishes the results of the latest in vitro tests which show that when present in the cell membrane, omega 3 fatty acids aid protect versus breast cancer development. The study recommends adding cold water fish to your diet, as they are exceedingly rich in DHA and EPA, the most indispensable omega 3 fatty acids.

Tests performed on lab rats antecedently implanted with breast cancer and which were given rich omega 3 aliments (fish oil) showed that their tumors have been scaled down by as much as 20 to 25%.

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Studying anatomy and physiology may be very frustrating. Have you ever searched for a more elementary way to study anatomy? I went indepth the pain of memorizing mountains of medical terms. I ought to tell you it’s not a simple task to do not forget them even English not to mention the Latin terms. If you genuinely want to make your study requiring little effort then remunerate close attention to this article and it will make you one step close to your goal.

o First of all memorizing the term and it is meaning is very similar to learning a alien language. That’s why I have borrowed a tip to do not forget the words. Take a bunch of index cards and write down a term on one side of the card and it is meaning on other side of it. Do it with all the terms you need to memorize. Now going through the cards one by one flipping them makes it much posing no difficulty to memorize the terms. Alternate back and forth until you may give the meaning or the term and then look at the definition original and name the term.

o Second tip that was exceedingly helpful for me is to have a lot of visualization of the humane anatomy. You know that persons receive the huge share of info from environs through eyes. Pictures and diagrams are good help. But I found even better solution. There are a great deal of pieces of software that show the three dimensional humane skeletal and muscular structure. Never underestimate the power of animation. You will be astonished how more comfortable it makes memorizing the bones and muscles of humane body.

Chart Of Human Anatomy

Classic illustrations by Peter Bachin. Shows anterior and posterior views of the muscular system. Also illustrates right half of the diaphragm, muscles of the posterior abdominal wall, and muscles of the right and right foot.Size is 20″ W by 26″ H.

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The chart is precisely like the one I saw in my classes at school! The detail is perfective and easy to see and reference to- the price is perfective too!

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This was a gift for my nephew who wants to be a doctor. It’s a great way for him to get intimate with the humane muscular system. Package came fine.

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This would be a beauteous product if it were shipped properly. Unfortunately, the laminated chart that we received was seriously damaged and can’t be repaired. The packaging we received was VERY ill-suited to the product. The chart was rolled in a 24″ x 5″ x 5″ light-weight, square-sided, cardboard box. This is a VERY weak geometric shape and a structure that is closely guaranteed to be crushed in shipment as it is fundamentally a light-weight box of air surrounded by paper being shipped in a truck filled with heavy boxes weighing up to 150 pounds. This is precisely what happened, the box was crushed and the quality chart which we paid for was turned into garbage. We’ve asked the marketer to switch their shipping protocol to structurally more inviolable tubes and ship a replacement. We’ll update this review after receiving feedback from the seller.

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In today’s newsletter, we’re going to talk regarding the vascular scheme — your arteries and veins. Unlike our discussion of the heart, which required a great deal of anatomy, our discussion of anatomy today will be much simpler. As I’ve stated previously, my goal in this series is not to make you doctors, but to aid you understand sufficient with regards to your body’s systems and how they work so that you may commune with your doctor and actively participate in your treatment. If you have high blood pressure, blood clots, or atherosclerosis, it’s of the utmost importance that you entirely grasp how that happened, the physiological aftermaths of any medical treatments, and any viable number of things from which only one can be chosen that might be available to you.

That’s what we will cover today.

Circulatory Systems

As we discussed previously, you have various distinct circulative systems.

The pulmonary system that carries deoxygenated blood away from the heart to the lungs, and then returns the refreshed oxygenated blood back to the heart.

The systemic scheme that carries the oxygenated blood away from the heart out to each single cell in your body, and then returns the expended deoxygenated blood back to the heart so that it may be sent out through the pulmonary system.

There is in truth a third system, the portal system, which loops within sure organs or areas of the body that we will talk about in future newsletters.

The essential thing to comprehend when it comes to these circulative schemes is that they are “closed looped.” Unless there is injury, no blood leaves them. As you will see, even the nourishment that each single cell in your body receives from your blood happens without that blood ever leaving the closed system. This becomes key when we talk with regards to blood pressure.

The circulative systems are comprised of:

- Arteries.

- Arterioles.

- Capillaries.

- Veins.

All told, these four parts make up some 50,000 miles of passageways in the body. Let’s take a look at them in more detail.

Arterial system

Arteries, arterioles, and capillaries make up the arterial system. Arteries and arterioles have only one function–to move blood allround the body. That’s all they do. They are channels, tubes, pipes if you will. As long as they are unclogged, flexible, and undamaged, they do their job. The necessary divergence amidst arteries and arterioles is one of size. Arterioles are just the smallest arteries you may see with the naked eye. Again, arteries and arterioles have only one function, to move blood. They do not feed any cells of the body–not even their own. That’s genuinely a fun little bit of trivia. The arteries of your body are not fed by the blood that flows through them. They require their own network of blood vessels called the vasa vasorum (literally, vessels of a vessel) that feed them — from the outside!

As I mentioned, I’m not going to get into naming all of the arteries in the body; but for the most part, arteries take their names from either the organs they supply (e.g.., the hepatic artery, which feeds the liver) or the areas through which they travel (e.g., the subclavian artery, which travels underneath the clavicle–AKA, the collar bone).

Capillaries

Capillaries are rather dissimilar in function. They are not designed to shuttle blood. In fact, blood scarcely flows through them at all as they are so little they concede only one blood cell at a time to pass through. Instead, the capillaries are the end point of the arterial system. It is in the capillaries that feed and oxygen are interchanged with each cell in your body (except your cornea and the lens of your eye). Amazingly, of the 50,000 miles of circulation in the body, capillaries comprise over 49,000 miles.

Unlike the arteries, capillaries are invisible to the naked eye. They are littler than a humane hair–microscopic. And it is because they are so little and their walls are so thin, that capillaries serve as the interchange scheme for feed and oxygen in the body. Keep in mind that each single cell in the body (except the cornea and lens) is near a capillary. That means that as blood passes through the ultra thin capillaries, it is easy for oxygen and tiny sugar and protein molecules (the end productions of digestion) to “exchange” through the walls of the vessel and feed each single cell in the body.

Capillaries also serve as the connecting point amid the arterial scheme and venous scheme that returns deoxygenated blood to the heart. The same interchange system that works to feed the cells of the body works in reverse. Cells pass their waste such as carbon dioxide back through the walls of the capillaries, where the blood cells not so long ago relieved of their oxygen payload, may now pick up the CO2 waste from the cell and carry it back to the lungs for interchange with fresh oxygen.

Surprisingly, there’s more “space” inside the tiny capillaries than may be filled by your entire blood supply. If all your capillaries were “open” simultaneously, your blood pressure would drop precipitously, and you would die. What happens, though, is that your body intelligently shunts blood into dissimilar capillaries as needed. When functioning properly, this is a pressure regulating mechanism. The body may open more capillaries to lower pressure, and close off sections if necessitated to raise pressure.

Note: our bodies retain the capacity to sprout new capillaries all around our entire lives.

Venous System

The venous scheme returns deoxygenated blood to the heart, and for the most part, it pretty much parallels the arterial scheme in all aspects–just in reverse. Whereas the arteries commence out big (the aorta) and end little (the capillaries), the venous scheme starts little (the capillaries) and ends big (the vena cava). Veins tend to run right next to their matching arteries, and in fact have similar names. The subclavian vein, for example, runs in tandem with the subclavian artery beneath your collar bone. The primary exception is the vena cava, which is the aorta’s counterpart.

How arteries and veins are constructed

In this section, we get started learning how difficulties occur. For it is their dissimilar construction (dictated by their dissimilar functions) that defines the nature of the things that may go wrong such as hardening of the arteries, high blood pressure, and blood clots.

Arteries

Arterial walls are composed of elastic tissue and smooth muscle. It is their elastic nature and the presence of substantial muscle tissue that allows them to exaggerate and contract as the heart beats. This allows them to even out the increase in pressure caused by each beat. This is one of the essential reasons why hardening of the arteries (atherosclerosis) increments blood pressure. If you pump more liquid through the same sized tube, pressure must increase. On the other hand, if the tube is flexible and may widen, the increase is less. (We will talk more with regards to this later.)

Veins

Veins are thinner walled than arteries and have less elastic tissue, and much, much less smooth muscle tissue. Instead, veins make use of valves and the muscle contraction of your body’s major skeletal muscles to squeeze blood along. This is the reason you’re asked to get up and walk around on a long plane flight–to prevent blood from pooling in your legs. As a side note, the lack of muscle in the walls of veins makes them more susceptible to bleeding when injured since there’s no muscle to clamp down.

Problems that may occur in arteries

There isn’t much mystery as to what the problem is–the build up of arterial plaque on the walls of the arteries and arterioles. There is, however, a great deal of mystery as to what causes it.

The basic problem is that arterial plaque (a combining of protein, calcium and cholesterol) starts building up on the walls of the arteries. This causes the arteries to both harden and narrow. So far so good! But what causes that buildup?

The cholesterol theory

The primary theory lays the blame on cholesterol–that as cholesterol levels climb in the blood, this causes plaque to form on the walls of the arteries. But this theory begins to collapse under even the most elementary scrutiny. As I brought up in my newsletter, the Cholesterol Myth, one of my favored questions to ask doctors is, “If cholesterol is the main culprit in heart disease, why don’t veins ever get narrowed and blocked?” And if you wanted to, you could throw capillaries into the equation too. Capillaries do not proof the build up of arterial plaque. (They do, however, clog with amyloid plaque in the brain. But that’s a dissimilar problem that we’ll cover in a later newsletter.)

Think when it comes to this for a moment. If you have cholesterol circulating evenly through the entire circulative system, but it only causes plaque to build up in the arteries and arterioles, not the capillaries or veins, then how may cholesterol be the indispensable cause of the problem? If cholesterol caused plaque to form, wouldn’t it form everywhere? Since it only forms in the arteries, doesn’t the problem have to be something distinguishable to those arteries?

The arterial wall theory

A more sophisticated version of the theory says that the build up of plaque is triggered by harm to the arterial wall–the endothelial lining. The lining comprises of a thin layer of endothelial cells that performs two critical functions:

- It protects the “innards” of the artery from toxic substances in the blood.

- It helps regulate the elaboration and contraction of the arteries by freeing a bio-chemical (cyclic GMP) into the cells of the smooth muscle in the arterial wall that modify the tone or firmness of the artery.

- In an try to repair harm to the endothelium, your body will “patch” the harm with plaque.

- This formulates one of two conditions–two sides of the same coin really.

Artherosclerosis (hardening of the arteries)

Damage to the endothelial lining is “managed” by the smooth muscle cells surrounding the lining. Smooth muscle cells respond to endothelial injury by speedily multiplying and fabricating a fibrin/calcium/cholesterol patch. These patches, called plaques occur just inside the lining and thicken the artery’s inner wall. Over time, given multiple injuries, the wall of the artery begins to harden and become dysfunctional, no longer expanding and contracting to regulate blood pressure –and regularly narrowing the passageway through which blood flows.

Arteriosclerosis (plaque build up)

Another way of describing this procedure is that your body brings about plaque to “paste over” any damaged areas–like a scab over a cut. Over time, given repeated injury, these plaques intrude more and more on the inner passage of the artery regularly compromising the capacity of the artery to exaggerate and contract and for blood to flow freely.

But it gets worse

The harm to the arterial wall also triggers an immune response with white blood cells flooding the area. This leads to a chronic inflammatory response in the blood vessel. Continued inflammation causes even more damage, which accelerates the process.

All of this, of course, brings up the $64,000 question: “Since the entire theory hinges on harm to the endothelial lining, what actually causes the harm to the lining, and why doesn’t it occur to the lining of the veins?”

Once again, oxidized fats and LDL cholesterol are named as the key culprits. Other suspected culprits include:

- Free radicals.

- High blood pressure (yes, high blood pressure begets more high blood pressure).

- Diabetes.

- High homocysteine levels.

- High C-Reactive Protein levels.

- Low levels of vitamin C (similar to scurvy).

- Low levels of nitric oxide.

- Heavy metals.

- Aging.

- Muscle matters

But once again, the question arises: “Are not all of these things present in the capillaries and veins too?” The answer, of course, is yes they are–which means there’s still a missing piece in the equation. The answer, according to the pH theory, lies not in what flows through the arteries and veins (which is identical), but in their construction (which is different). The key divergence amidst arteries and veins is in the amount of muscle tissue surrounding the endothelial lining. In arteries and arterioles, the smooth muscle is extensive. In veins, it is minimal. And in capillaries, it is completely absent. Why does this matter?

It matters because when muscle tissue is used it develops lactic acid. If your body is healthful (in an alkaline state) and has ready access to an plentiful source of oxygen rich blood, that lactic acid may clear quickly. But for those humans who eat a high acid forming diet and are in an acidic state, the lactic acid can not clear quickly. (Remember, blood vessels do not have direct access to the oxygen in the blood that flows through them. They are dependent on the vasa vasorum.) It is the lactic acid that provides the final trigger that causes harm to occur in arterial linings, but not so in veins. It is the presence of assembled lactic acid in the smooth muscles surrounding arteries that at long last causes plaques to form.

But even beyond lactic acid, there’s another area where muscle tissue matters: nitric oxide. The contraction of the muscles in the arterial walls is regulated by a signaling molecule that we referred to earlier called cyclic guanosine monophosphate (cyclic GMP) in the muscle cells. Cyclic GMP causes the arterial muscle to relax, in preparation for it is next contraction. Cyclic GMP is triggered by nitric oxide, which is developed in the endothelial lining. The capacity of the lining to invent sufficient nitric oxide to maintain artery dilation is one of it is most important functions. As harm proceeds to build in the lining, it blocks nitric oxide-induced dilation, therefore stiffening the arteries.

High Blood Pressure

If the arterial blockages take place in your coronary arteries, the result, as we’ve discussed previously, is coronary heart sickness and a heart attack. If it happens in the carotid arteries leading to the brain, it may cause a stroke.

In most cases, however, the harm happens systemically, allround your arterial system, and the result is high blood pressure. As a quick review, blood pressure is a measurement of the two pressures in your circulative system as your heart beats. The increased pressure invented in your circulative scheme by the contraction of the left ventricle is referred to as systolic pressure. The scaled down pressure for the duration of relaxation is called diastolic pressure. These are the two numbers your doctor gives you when reading your blood pressure (e.g., 120 over 70). Both low and high blood pressure are dangerous, but low blood pressure is ordinarily having little impact to manage. High blood pressure, on the other hand, have a tendancy to be more intractable and harder to manage–and accordingly more dangerous.

Your body has a heap of mechanisms for controlling blood pressure.

- It may modify the amount of blood the heart pumps.

- It may modify the diameter of arteries, and the volume of blood in the bloodstream.

- To increase blood pressure, it may pump more blood by pumping more forcefully or more rapidly.

- It may likewise increase pressure by narrowing arteries (particularly the arterioles), forcing the blood from each heartbeat through a narrower space than normal.

- It may seal off capillaries forcing the blood into a littler space, thereby increasing pressure.

- The body may add liquid to the bloodstream (regulated by the kidneys) to increase blood volume and thence increase blood pressure.

- And it may remove liquid from the blood (also regulated by the kidneys), thereby decreasing pressure.

All of these things take place automatically, regulated by a healthful body, without your even thinking regarding it. In addition, blood-pressure measurements may vary allround the day, affected by everything from:

- Food.

- Alcohol.

- Caffeine.

- Smoking.

- Stress.

- Climate.

- And the time of day.

Blood pressure changes that take place naturally for the duration of the day are the result of the body’s internal (circadian) rhythms. In most people, blood pressure rises quickly in the early morning hours, in anticipation of rising and beginning the day. This is not the result of the physical act of rising but is a preset system that mechanically increments a person’s blood pressure at that time. Likewise, pressure ordinarily starts dropping early in the evening in anticipation of going to sleep.

All of these things noted so far, have not one thing to do with clinical hypertension unless they result in secondary harm such as may be caused by smoking and alcohol or sustained stress. Clinical hypertension is a chronic and dangerous condition caused by:

- Constricted arteries.

- Hardened arteries.

- Malfunctioning kidneys (which we’ll talk regarding in a subsequent newsletter).

If left untreated, chronic hypertension may cause:

- Damage to the heart muscle because of the extra load it puts on the heart.

- Strokes.

- Kidney damage–which leads to more hypertension, which leads to more kidney damage, etc.

And ultimately, it kills you.

Problems that may occur in veins

As we’ve already discussed, veins do not have a substantial amount of muscle tissue to contract and squeeze blood along. That means that without physical action to cause the skeletal muscles to squeeze the veins:

- Blood has a tendency to pool and stop flowing in veins–particularly in the legs where gravity works versus you.

- Blood that isn’t flowing have a tendancy to clot.

- Clots tend to propagate more clotting around the basi clot.

- Cumulatively, this may form very big clots.

- Large clots that stay in place and block the flow of blood cause phlebitis.

If the clot breaks free and starts journeying through the circulative system, it’s called a thrombus. At whatsoever point it lodges in a blood vessel and blocks it, it’s called an embolism. If you think back to our discussion of the venous system, you’ll do not forget that veins get steadily more prominent as blood moves back to the heart. That means that clots that break free in the legs are improbable to be stopped anyplace on their way back to the heart. The introductory place they are likely to lodge is when the right ventricle of the heart pumps them out into the pulmonary circulative scheme on the way to the lungs. If the clot is reasonably small, it will lodge in the lung itself and block the flow of blood to a division of the lung, killing it. This is called a pulmonary embolism. Larger clots may in truth lodge in the pulmonary artery feeding an entire lung…killing the lung just like that. Or the clot may lodge at the juncture where the pulmonary artery divides amidst the two lungs, which will kill both lungs simultaneously…in an instant.

DVT, or deep vein thrombosis, is the term now commonly related with clots that form as the result of prolonged sitting on an airplane. They tend to break free the next time you begin moving again with any vigor. This may be various days or weeks after the plane flight itself, which means a great deal of people never connect the two events.

There is one other remarkable place that clots tend to form. As a result of low blood flow or damaged valves, clots may form in the left atrium of the heart. If the clot forms there, it’s already past the pulmonary circulative scheme so it can’t affect the lungs. Unfortunately, the next stop for the clot is out into the systemic circulative system, where it has a good prospect of being pushed up into the brain causing a stroke.

What doctors do when it comes to these problems

Medical treatments for vascular difficulties never address the actual causes, but seek rather to strength test results back into line. What is your doctor likely to offer?

Clogged arteries

Modern medicine in truth only has two approaches.

1. Surgically repair the damaged area (bypasses and angioplasties).

2. Use drugs to improve the flow of blood through the damaged area and denigrate the production of cholesterol, which serves as one of the triggers.

Neither of these approaches, of course, actually deals with the real problem.

High blood pressure

When it comes to high blood pressure, doctors rely closely wholly on pharmaceutical drugs. The four major classes of drugs are:

1. Diuretics, which reduce pressure by making you pee out water from your body. Reduce the volume of liquid in your blood, and you reduce the pressure. Unfortunately, side effects may include dizziness, weakness, an increased peril of strokes, and impotence. (Not to worry, there are medications to alleviate the side effects.)

2. Calcium channel blockers, which work to relax and widen the arteries–thus reducing blood pressure. Then again, a major side effect of channel blockers is a 60% increased risk of heart attack.

3. Beta blockers, which work by weakening the heart so it won’t pump as strongly, thereby reducing blood pressure. One of the major troubles with beta blockers, though, is the increased danger of congestive heart failure.

4. ACE inhibitors (the new drugs of choice), which like the calcium channel blockers, likewise work to relax and widen the arteries. Unfortunately, ACE inhibitors may create severe allergic reactions, may be deadly to fetuses and children who are breastfeeding, and may cause severe kidney damage.

Again, none of these drugs deals with the actual cause of the high blood pressure. They are merely an try to strength test numbers into line and prevent humans from without delay dying.

Blood clots and DVT

If doctors are worried when it comes to clots (such as after bypass surgery), they put people who are in need of medical care on blood thinners. The popular is Coumadin (warfarin). Aside from the ordinary jokes that Coumadin is fundamentally rat poison (which it is), it has severe side effects. It may cause severe internal bleeding that may be life-threatening and even cause death. You may always tell a person on warfarin by the extensive bruising all over their body since even the slightest bump or touch is sufficient to cause internal bleeding. It’s a bit like using dynamite to open a locked door. It may do the job, but you need to be oh so careful or you’ll blow up the building at the same time. There are better choices.

Note: a lot of people might think aspirin is a good alternative. It’s not. While aspirin may be beneficial at keeping blood flowing through arteries, studies indicate it has no effect on preventing clots from forming in veins.

What are the options?

As it turns out, for most major heart problems, you have a world of alternatives–certainly safer and many times far more effective than their medical counterparts.

Clogged arteries

- Studies have shown that dietary changes alone may unplug arteries.

- Proteolytic enzymes, particularly formulas that incorporate either nattokinase or lumbrokinase, may break down the proteins that hold plaque together stuck to arterial walls–effectively dissolving it.

Proteolytic enzymes may likewise help dissolve scarring of the endothelial lining.

And proteolytic formulas that incorporate seaprose-s, serrapeptase, and/or endonase may help reduce arterial inflammation that both constricts arteries in real time and contributes to future long term damage.

- Sufficient Omega-3 fatty acids in the diet also support reduce arterial inflammation and dramatically reduce the circulating levels of detrimental NEFAs.

- Antioxidants such as SOD, pomegranate, grape seed extract (AKA OPCs), and pycnogenol help heal the endothelial lining, thereby preventing future plaque and helping to heal current plaque.

- Methylating supplements such as B6, folic acid, B12, TMG, and SAMe support reduce homocysteine levels, thereby reducing harm to the endothelial lining.

- L-arginine and noni extract support the smooth muscle in arterial walls in obtaining sufficient nitric oxide to function properly.

- Regular heavy metal detoxing may reduce a major cause of botheration to the endothelial lining and a essential instigator of plaque formation.

- And raising body pH through proper diet and the use of supplements such as coral calcium reduces lactic acid levels in the arterial smooth muscle, thereby minimizing harm to arterial linings.

As you may see, there is a world of selections you may make that may dramatically alter your vascular outcomes. Virtually all of them are covered if you’re following the Baseline of Health Program.

High blood pressure

Pretty much everything you do to reduce clogging of the arteries will, by definition, aid to reduce blood pressure. In addition, though, you may likewise consider:

Lose weight. Simple laws of physics implement here. As we’ve already discussed, your blood vessels have to service each single cell in your body. The more body mass you have, the more pressure you need to strength blood through the system. Lose weight; less pressure required.

If you smoke, stop. Smoking constricts blood vessels and raises pressure.

If you’re stressed, undertake meditation or biofeedback. As share of your body’s “flight and fight” mechanisms, stress increments heart rate and blood pressure to aid respond to the short term stress of an attack from a saber toothed tiger. Twenty-four/seven stress was not designed into the system. Prolonged stress unquestionably impacts blood pressure levels. Even if you have clogged arteries, reducing stress levels may still support drop your blood pressure levels significantly.

Herbs such as passionflower, apocynum venetum, hawthorne, and stevia (yes stevia) have all been shown in clinical studies to aid lower blood pressure.

Blood clots and DVT

Proteolytic enzymes, peculiarly formulas that comprise either nattokinase or lumbrokinase are just as effective at preventing clots, with wide ranging dosage tolerances. In other words, good proteolytic formulas work with minimal probability of side effects. In fact, a good systemic proteolytic enzyme formula that also holds enzymes such as endonase, seaprose-s, or serrapeptase may have multiple beneficial effects for the circulative system in addition to reducing clotting. Such formulas may play a major role in reducing inflammation and scarring in the cardiovascular system and heighten cardio perfomance in athletes.

Conclusion

When it comes to most forms of heart sickness related with the arteries and veins, you have a world of alternatives–certainly safer and many times far more effective than their medical counterparts. It’s likewise worth noting again that if you are following the Baseline of Health Program, then you’re already doing most of them.

Which brings us to the final percentage of our series on the anatomy, physiology, and impairment of normal physiological functions of the cardiovascular system–your blood. In the next issue will take on this most complex of subjects.

Real Heart Diagram

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Find the concise, practical business info you need right now in the Architect’s Essentials of Professional Practice Series. These authorized guides quickly make you an instant expert on the best business exercises crucial for success in today’s design and construction professions. Each portable, affordable, user-friendly volume gives you:

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For design firms that want to take control of their syndication plans and increase business, Architect’s Essentials of Marketing is the single-source guide with all the answers. Through concise, step-by-step instructions, it illuminates all distinct features of creating a winning merchandising system and covers how to leverage a assortment of retail tools and resources. Valuable, real-world guidance from an industry-leading marketing professional addresses how to manage the business development cycle to acquire clients and build your business.

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About the AuthorDAVID KOREN is Marketing Director for Gensler’s 250-person New York office where he is responsible for selling strategy, strategic alliances, market research, and public relations. Koren is co-chair of the marketing committee of the AIA New York chapter and is an active fellow member of the Society for Marketing Professional Services.

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